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Epiphone 03gp5s owner manual.Apoptotic cell death is an important pathway for limiting post-embryonic development and tissue homeostasis, a role clearly demonstrated by mutations in the pro-apoptotic gene Bax. Accumulating evidence also suggests the involvement of apoptosis in the normal development of diverse tissues, including brain, liver, heart, kidney and pancreas. In the nervous system, apoptosis contributes to the formation of many cells types, including neurons, oligodendrocytes and astrocytes. Loss of post-embryonic cell death in Drosophila results in neuromuscular dysfunction and other abnormalities. Therefore, an understanding of the molecular and genetic events that determine cell death is likely to provide crucial insights into the fundamental biology of the nervous system and lead to the rational treatment of neurological diseases. Our previous studies indicate that the Drosophila Bcl-2 homolog Dark promotes cell death in embryos, pupal wings and adult legs. To understand the molecular basis for Dark’s function, Dark homologs have been identified from several Drosophila species, and we have recently cloned the Drosophila homolog of mammalian Bcl-2. Over-expression of Dark blocks cell death induced by a number of stimuli, including gamma-irradiation, heat shock and expression of the pro-apoptotic gene hid. Our studies indicate that Dark blocks cell death by inhibiting the apoptotic cascade. Dark also acts in vivo to protect the developing eye from radiation-induced cell death. The fact that no homologs of the Death domain-containing proteins of the Bcl-2 family have been found in any higher organism suggest that Dark is likely to be the founding member of a novel class of cell death regulators. However, it is not clear whether Dark is sufficient to explain the various forms of cell death observed in Drosophila or whether, as has